Traumatic Cardiac Arrest Isn’t Hopeless but it is Time-Critical
- ROCKET PR
- 11 minutes ago
- 4 min read
Author: Kaleb Lachenicht
Transforming EM through patient safety systems, Just Culture development, and clinical excellence! Passionate about connection, building systems, and developing teams.

For years, traumatic cardiac arrest (TCA) has been treated as a near-futile endpoint. And in a lot of ways, especially in the South African environment, it is still is. Long response times, resource limitations (ambulances especially in rural spaces are few and far between).
But some contemporary data, and modern trauma systems, tell a slightly different story.
Survival is possible and good neurological outcomes do occur. The difference is rarely luck... it’s timing, prioritisation, and systems.
This narrative review by Carenzo et al. reframes TCA not as a single event, but as a dynamic physiological spectrum, and that framing matters at the bedside, in the back of the aircraft, and in the emergency centre.
Traumatic Cardiac Arrest Is Not One Thing
One of the most useful contributions of this review is the reminder that “TCA” is an umbrella term.
The authors distinguish between:
LOST (Low-Output State in Trauma)
NOST (No-Output State in Trauma)
Why does this matter?
Because LOST has survival potential, if we can get to the patient rapidly and bring the interventions that might change the outcome to them.
NOST, especially after prolonged downtime, is almost never recoverable.
Peri-arrest...that grey zone just before collapse, is where our interventions matter most. Recognising we are heading in that direction is often the most important start.
HOTT Is Necessary... But its NOT ENOUGH
Most of us are fluent in HOTT:
Hypovolaemia
Oxygenation (hypoxia)
Tension pneumothorax
Tamponade
The review reinforces a key principle:
These must be treated simultaneously, not sequentially.
But it also pushes us further.
A contemporary approach adds a second layer:
Metabolic failure (especially hyperkalaemia)
Haemostatic collapse
Coronary perfusion pressure as the true target
This explains why chest compressions often fail in TCA, not because CPR is “bad”, but because there is no preload, no coronary flow, and no physiology to support it.
In hypovolaemic or obstructive arrest, restoring circulation beats compressing an empty heart.
Chest Compressions: Context Matters
This paper challenges dogma in a nuanced way, and the nuance is ALWAYS where the magic is for me...
In exsanguination or tamponade, chest compressions alone do little
In isolated TBI, asphyxia, or unclear aetiology, they may still matter
If you have the manpower to do both, treat causes and compress, do both
The key message isn’t “don’t do CPR”. It’s don’t let CPR delay definitive action.
Penetrating vs Blunt Trauma: Different Games, Different Odds
The outcomes are stark:
Penetrating TCA: ~10% survival
Blunt TCA: ~2–3% survival
Why?
Penetrating injuries are often:
Single-system
Surgically correctable
Temporally reversible
Blunt trauma is usually multisystem, diffuse, and metabolically catastrophic.
This distinction should influence:
Decision-making
Aggressiveness of intervention
Team expectations and psychological framing
Advanced Interventions: Timing Is Everything
Resuscitative Thoracotomy (RT)
RT is not heroic, it’s physiology-driven. When used for the right patient at the right time and with the right intention, it
Indications are narrow but powerful:
Penetrating chest trauma
Signs of life
Short downtime
When tamponade is present, RT is the treatment, not an option.
REBOA
REBOA has shifted the landscape for sub-diaphragmatic haemorrhage.
Key takeaways:
Zone 1 for TCA
Early femoral access matters
It’s a bridge — not a solution
Importantly, REBOA does not replace RT in penetrating thoracic trauma — and using the wrong tool at the wrong time can worsen outcomes.
The Forgotten Killers: Potassium and Calcium
One of the most under-appreciated sections of this review is metabolic failure.
Hyperkalaemia occurs early in haemorrhagic shock
It correlates strongly with failure to achieve ROSC
Massive transfusion worsens it
Aggressive correction — calcium, insulin, glucose — may be the difference between electrical silence and meaningful cardiac activity.
Similarly, hypocalcaemia:
Impairs coagulation
Reduces myocardial contractility
Worsens shock
This is advanced resuscitation thinking — and it belongs in retrieval medicine.
Systems Matter More Than Any Single Skill
The authors are clear about this: outcomes improve when advanced care happens early and on scene, delivered by trained teams within mature systems.
This isn’t about individual heroics. Survival is also linked to the THINKING PRACTITIONER - we cant expect our teams to operate like robots, using abnormal algorithms to guide each step and never engaging our brains.... We need to ask more of our providers and teams if we want to see better survival from the cardiac arrest trauma patient
It’s about:
Algorithms
Training
Governance
Rehearsed decision-making under pressure
TCA survival is a system achievement.
Bottom Line
Traumatic cardiac arrest is not futile, but it is unforgiving
Treat causes before rhythms
HOTT plus metabolic control is the modern approach
USE YOUR BRAIN - and treat the patient not the algorithm
Chest compressions are contextual, not automatic
RT and REBOA demand clarity, timing, and training (obviously in the prehospital space in SA this still a way off for most patients and in most towns)
Survival lives in the peri-arrest window... not after prolonged arrest. Catch the deterioration early and treat it aggressively.
Over to you
How does your service approach traumatic peri-arrest?
Where do you think the biggest gains still lie, training, access to interventions, or decision-making under pressure?
Reference
Carenzo, L. et al. (2024). Contemporary management of traumatic cardiac arrest and peri-arrest states: a narrative review. Journal of Anesthesia, Analgesia and Critical Care, 4:66.


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